Oura Ring Gen 4 sensor data, not clinical measurementsN=1 case study, not validated for clinical decisionsHEV diagnosed Mar 18; Day 109 post-ruxolitinibMore
Consumer wearable data can support exploratory review only. The HEV diagnosis, temporally confounded with treatment start, remains a material confounder.

Breathing Rate Analysis & BOS Screening

Module 6: Comparative Breathing Analysis
P1 MEAN BREATH
In range
14.1brpm
Range: 12.0-20.0 brpm normal
Average nighttime breathing rate
P2 MEAN BREATH
In range
15.4brpm
Range: 12.0-20.0 brpm normal
Average nighttime breathing rate
P3 MEAN BREATH
In range
16.6brpm
Range: 12.0-20.0 brpm normal
Average nighttime breathing rate
P1 TREND
Stable
Stable
Slope: 0.0052/day
BOS RISK
Low Risk
Low Risk
0.0% nights above 18.0 brpm
Bronchiolitis Obliterans Syndrome screening
DISTRIBUTION

Distribution Comparison

Mann-Whitney U: p<0.001 · Cohen's d: -2.83 (large) · 95% CI for mean difference: (-1.52, -1.18) brpm

RUX EFFECT

Ruxolitinib Effect on Breathing Rate

COUPLING

Respiratory-Autonomic Coupling

Respiratory sinus arrhythmia (RSA): In healthy individuals, higher HRV correlates with lower breathing rate through vagal modulation. Disrupted coupling may indicate autonomic dysfunction.

Patient 1: r=0.10, p=0.342, n=88 -- Coupling disrupted/weak
Patient 2: r=0.01, p=0.900, n=518 -- Coupling disrupted/weak
Patient 3: r=-0.44, p=0.043, n=22 -- Coupling intact

BOS SCREENING

BOS Screening Panel

CLINICAL INTERPRETATION

Clinical Interpretation

BOS Screening: P1's breathing rate trend is classified as Low Risk. The linear trend slope is +0.0052 brpm/day (0.0% of nights above the 18.0 brpm elevated threshold). Post-HSCT patients require ongoing monitoring for BOS, especially with chronic GVHD.

Ruxolitinib Effect: Statistically significant difference in breathing rate pre vs post Ruxolitinib (delta: +0.51 brpm, effect: medium). Ruxolitinib, a JAK inhibitor, reduces inflammatory cytokines and may modulate respiratory drive indirectly through GVHD suppression.

Autonomic Coupling: Respiratory-autonomic coupling is disrupted or weak in Patient 1 (Spearman r=0.10). Disrupted breath-HRV coupling in HSCT patients may reflect autonomic neuropathy or chronic inflammatory burden affecting vagal tone.

Anomalous Nights (Patient 1)

Nights with breathing rate >2 SD above personal mean (3 detected):

DateBreath (brpm)Z-ScoreHRV (ms)HR (bpm)
2026-01-2015.92.35.096.5
2026-01-2215.82.18.091.625
2026-04-1116.02.512.074.625

This analysis uses nighttime breathing rate from Oura Ring sleep periods (long sleep only). Clinical BOS diagnosis requires pulmonary function tests (FEV1, DLCO). Wearable breathing rate is a screening adjunct, not diagnostic.

METHODOLOGY

Methodology

Data Source: Oura Ring sleep periods (type=long_sleep), average_breath column. Both patients' data loaded from separate SQLite databases.

BOS Screening: Linear regression on daily breathing rate. Thresholds: elevated >18.0 brpm, concerning trend >0.02 brpm/day. Normal sleep breathing range: 12.0-20.0 brpm.

Ruxolitinib Comparison: Mann-Whitney U test (non-parametric, appropriate for non-normal distributions). Cohen's d for effect size, bootstrap 95% CI for mean difference (10,000 iterations).

Cross-Patient Comparison: Mann-Whitney U with Cohen's d. Z-score normalization relative to each patient's own mean/SD for fair comparison.

Coupling Analysis: Spearman rank correlation between breathing rate and HRV (RMSSD) on overlapping dates. Respiratory sinus arrhythmia predicts negative correlation (higher HRV, lower breath rate) when vagal modulation is intact.

Anomaly Detection: Nights with breathing rate >2.0 SD above personal mean are flagged. Co-occurring HRV and HR values provide clinical context.